Chemo- and optogenetic activation of hypothalamic Foxb1-expressing neurons and their terminal endings in the rostral-dorsolateral PAG leads to tachypnea, bradycardia, and immobility.
Reto B. Cola, Diana M. Roccaro-Waldmeyer, Samara Naim, Alexandre Babalian, Petra Seebeck, Gonzalo Álvarez-Bolado, Marco R. Celio
The paper uses optogenetic and chemogenetic tools to activate Foxb1-expressing neurons in the hypothalamus and their terminals in the PAG, revealing their role in inducing tachypnea, bradycardia, and immobility.
eLife · Feb 1, 2024Peer-reviewedNeuroscience