Author

Carmen Romero‐Molina

Icahn School of Medicine at Mount SinaiORCID 0000-0002-3119-961XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Genetic & Genomic Medicine· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 4 Research Focus:differential expression· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Research Focus:benchmarking· 1

Frequent Co-Authors

Alison GoateiD· 3 shared Edoardo M. MarcoraiD· 2 shared Tulsi PateliD· 2 shared Wen Yi See· 2 shared Alberto PascualiD· 1 shared Alicia E. Rosales‐NievesiD· 1 shared Ana B. Muñoz‐ManchadoiD· 1 shared Ana María Muñoz-CabelloiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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3 shared papersNeuroscience

Alison GoateORCID

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4 papers

Recent·Most cited

NeurosciencebioRxivPreprintMar 26, 2026

Reduced LACTB expression in myeloid cells is associated with elevated succinylcarnitine levels and reduced Alzheimers disease risk.

Carmen Romero‐Molina, Ruben Gomez‐Gutierrez, Wen Yi See…·Alison Goate

The paper investigates the role of LACTB in Alzheimer's disease risk through Mendelian Randomization and experimental models, finding reduced LACTB expression lowers AD risk and increases succinylcarnitine levels.

DOI 10.64898/2026.03.24.711053

humanproteomics

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ImmunologyCell ReportsPeer-reviewedJun 25, 2025

Cytokine-induced reprogramming of human macrophages toward Alzheimer's disease-relevant molecular and cellular phenotypes in vitro

Anna Podlesny‐Drabiniok, Carmen Romero‐Molina, Tulsi Patel…·Alison Goate

This study reprograms human macrophages in vitro using cytokines to mimic Alzheimer's disease-relevant phenotypes, showing transcriptomic and functional changes similar to disease-associated microglia.

7 citations

humanbulk RNA-seq

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NeurosciencebioRxivPreprintOct 1, 2024

The PHD3-FOXO3 axis modulates the interferon type I response in microglia aggravating Alzheimer's disease progression

Manuel A. Sánchez-García, Nieves Lara-Ureña, Rosana March‐Díaz…·Alberto Pascual

This study identifies the PHD3-FOXO3 axis as a regulator of the interferon type I response in microglia, which contributes to Alzheimer's disease progression.

DOI 10.1101/2024.10.01.616066

mousehuman

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Genetic & Genomic MedicineAlzheimer s & DementiaPeer-reviewedOct 12, 2022

The Role of Mitochondrial genome abundance in Alzheimer's Disease

Nadia V. Harerimana, Devashi Paliwali, Carmen Romero‐Molina…·Shea J. Andrews

The paper investigates the association between mitochondrial genome abundance and Alzheimer's disease, finding lower abundance correlates with worse neuropathology and cognitive performance.

33 citations

humanDNA methylation

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