Author

Marina K Triplett

Columbia UniversityORCID 0000-0002-3547-2839Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Research Focus:perturbation response prediction· 1

Frequent Co-Authors

Livio PellizzoniiD· 2 shared Maria Jose CarliniiD· 2 shared Christopher E. HendersoniD· 1 shared Daniel Martin WattersoniD· 1 shared Eileen Workman· 1 shared Ivan TattoliiD· 1 shared Jorge Espinoza‐DeroutiD· 1 shared Meaghan Van AlstyneiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersNeuroscience

Livio PellizzoniORCID

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2 papers

Recent·Most cited

A chemical screen identifies p38 MAPK inhibition as a candidate neuroprotective strategy for combinatorial SMA therapy

Maria Jose Carlini, Jorge Espinoza‐Derout, Meaghan Van Alstyne, Sarah Tisdale, Eileen Workman, Marina K Triplett, Ivan Tattoli, Shubhi Yadav, Christopher E. Henderson, Daniel Martin Watterson, Livio Pellizzoni

This study identifies p38 MAPK inhibition as a neuroprotective strategy that synergizes with SMN upregulation to improve motor function and survival in SMA mice.

bioRxiv · Mar 6, 2025 · PreprintNeuroscience

Neuromuscular denervation and deafferentation but not motor neuron death are disease features in the Smn 2B/- mouse model of SMA

Maria Jose Carlini, Marina K Triplett, Livio Pellizzoni

This study investigates SMA pathology in Smn2B/- mice, finding that neuromuscular denervation and deafferentation, not motor neuron death, are consistent features of the disease.

PLoS ONE · Aug 1, 2022Peer-reviewedNeuroscience