Author

Sarah Tisdale

Columbia University Irving Medical Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:drug response / drug discovery· 1 Research Focus:perturbation response prediction· 1

Frequent Co-Authors

Livio PellizzoniiD· 3 shared Meaghan Van AlstyneiD· 3 shared Christian M. SimoniD· 2 shared Daniel Martin WattersoniD· 2 shared George Z. MentisiD· 2 shared Christopher E. HendersoniD· 1 shared Eileen Workman· 1 shared Elena BianchettiiD· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersNeuroscience

Livio PellizzoniORCID

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3 papers

Recent·Most cited

A chemical screen identifies p38 MAPK inhibition as a candidate neuroprotective strategy for combinatorial SMA therapy

Maria Jose Carlini, Jorge Espinoza‐Derout, Meaghan Van Alstyne, Sarah Tisdale, Eileen Workman, Marina K Triplett, Ivan Tattoli, Shubhi Yadav, Christopher E. Henderson, Daniel Martin Watterson, Livio Pellizzoni

This study identifies p38 MAPK inhibition as a neuroprotective strategy that synergizes with SMN upregulation to improve motor function and survival in SMA mice.

bioRxiv · Mar 6, 2025 · PreprintNeuroscience

SMN controls neuromuscular junction integrity through U7 snRNP

Sarah Tisdale, Meaghan Van Alstyne, Christian M. Simon, George Z. Mentis, Livio Pellizzoni

This study identifies a role for SMN in maintaining neuromuscular junction integrity through U7 snRNP function in SMA mice.

Cell Reports · Sep 1, 2022Peer-reviewedNeuroscience

Stasimon contributes to the loss of sensory synapses and motor neuron death in a mouse model of spinal muscular atrophy

Christian M. Simon, Meaghan Van Alstyne, Francesco Lotti, Elena Bianchetti, Sarah Tisdale, Daniel Martin Watterson, George Z. Mentis, Livio Pellizzoni

This study shows that Stasimon overexpression in SMA mice improves motor function by preventing synaptic loss and motor neuron death through mechanisms involving p53 regulation.

Cell Reports · Jan 1, 2019Peer-reviewedNeuroscience