R. Noah Perry

The paper identifies female-biased VSMC gene regulatory networks and highlights MYH9 as a key regulator of fibrous plaque phenotype in females.

The paper develops a computational model of cardiac proliferation signaling that identifies key regulators and predicts Hippo to cMyc module crosstalk driving cardiomyocyte proliferation.

The paper identifies dysregulated metabolic pathways in vascular smooth muscle cells during phenotypic switching and links them to atherosclerosis.

This study identifies FHL5 as a causal gene for vascular disease risk through eQTL colocalization and functional analyses, showing its role in vascular remodeling and calcification.

The paper identifies eQTLs and sQTLs in human aortic SMCs that colocalize with CAD GWAS loci, highlighting splicing regulation as a mechanism for CAD risk.