Author

Dillon Lue

University of VirginiaORCID 0000-0003-2008-702XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Organism:human· 3 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Mete CivelekiD· 2 shared Rédouane AherrahrouiD· 2 shared Arlene H. SharpeiD· 1 shared Arnav MehtaiD· 1 shared Aziz M. Al’KhafajiiD· 1 shared Clint L. MilleriD· 1 shared Doris WongiD· 1 shared Ernest Diez BenaventeiD· 1 shared

Activity by Year

2022-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersGenetics

Mete CivelekORCID

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3 papers

Recent·Most cited

ImmunologybioRxivPreprintJun 17, 2026

Discovery of Tcf7 regulators with clonally-resolved CRISPR screens identifies Trim28 as a mediator of CD8 T cell differentiation in tumors

Marc A. Schwartz, Dillon Lue, Mohammed Mutaher…·Nir Hacohen

The paper identifies Trim28 as a regulator of CD8 T cell differentiation in tumors using clonally-resolved CRISPR screens and shows its role in maintaining TCF7+ stem-like cells while impairing effector differentiation.

DOI 10.64898/2026.06.14.732162

humanscRNA-seq

GeneticsHuman Genetics and Genomics AdvancesPeer-reviewedNov 30, 2022

Genetic Regulation of Circular RNA Expression in Human Aortic Smooth Muscle Cells and Vascular Traits

Rédouane Aherrahrou, Dillon Lue, Mete Civelek

This study identifies circRNA expression patterns and genetic associations in human aortic smooth muscle cells and links them to cardiovascular traits.

10 citations

humanstatistical model

GeneticsbioRxivPreprintJan 24, 2022

Genetic regulation of human aortic smooth muscle cell gene expression and splicing predict causal coronary artery disease genes

Rédouane Aherrahrou, Dillon Lue, R. Noah Perry…·Mete Civelek

The paper identifies eQTLs and sQTLs in human aortic SMCs that colocalize with CAD GWAS loci, highlighting splicing regulation as a mechanism for CAD risk.

4 citations

humanbulk RNA-seq