Markus Philipp Radsak

This study identifies kinome activity and transcriptomic changes in human neutrophils after TREM-1 activation, revealing potential kinase targets for inflammatory and cancer therapies.

This study shows HDAC1 and HDAC2 protect BCR-ABL-positive CML cells from replication stress-induced apoptosis via the p73-NOXA axis.

This study shows that transient YAP/TAZ inhibition can suppress tumor growth in advanced cancers and enhances immunotherapy when combined with immune checkpoint blockade.

This study identifies granulocytes as the primary source of exercise-induced cfDNA, which can affect liquid biopsy accuracy in healthy individuals and patients with hematological malignancies.