William J. Ray

The paper investigates the role of LACTB in Alzheimer's disease risk through Mendelian Randomization and experimental models, finding reduced LACTB expression lowers AD risk and increases succinylcarnitine levels.

This study shows that DLK inhibition after spinal cord injury improves neuroprotection in female mice but worsens recovery and increases lesion size in males.

This study shows that pathogenic tau accelerates transposable element activation in the aging mouse brain, particularly ERVs, suggesting a link between tauopathy and transposable element-induced neurotoxicity.

The paper identifies Moesin as a key effector of tau-induced neurotoxicity by disrupting neuronal identity through aberrant cell cycle activation in Alzheimer's disease and tauopathies.

This study investigates the context-specific roles of DLK/p-c-Jun signaling in the cerebellum and hippocampus, showing distinct effects on synaptic maintenance and neuroinflammation.