Author

Julián Panés

Hospital Clínic de BarcelonaORCID 0000-0002-4971-6902Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Organism:human· 3 Research Focus:cell-cell communication· 2 Approach:experimental assay· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Organism:mouse· 1

Frequent Co-Authors

Ana Maria CorralizaiD· 3 shared Azucena SalasiD· 3 shared Agnes Fernandez-ClotetiD· 2 shared Angel GineriD· 2 shared Ángela Sanzo-MachucaiD· 2 shared Berta CaballoliD· 2 shared Elena RicartiD· 2 shared Elisa Melón-ArdanaziD· 2 shared

Activity by Year

2025-2025 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersImmunology

Ana Maria CorralizaORCID

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3 papers

Recent·Most cited

TL1A-activated T cells remodel the rectal mucosa in Crohn's disease patients with perianal fistulizing disease

Victòria Gudiño, Jae Won Cho, Berta Caballol, Ana Maria Corraliza, Marisol Veny, Isabella Dotti, Lívia Moreira Genaro, Ángela Sanzo-Machuca, Elisa Melón-Ardanaz, Maria Carme Masamunt, Míriam Esteller, Iris Teubel, Lisseth Robbins, Angel Giner, Cristina Prieto, , Azucena Salas

The paper identifies TL1A-driven cellular and transcriptomic changes in rectal mucosa of Crohn's disease patients with perianal fistulizing disease, revealing a novel TL1A-LT1{beta}2/IL-22 axis independent of TNF signaling.

bioRxiv · Jun 26, 2025 · PreprintImmunology

Understanding the mechanisms underlying the lack of response to Janus kinase inhibition in ulcerative colitis

Elisa Melón-Ardanaz, Marisol Veny, Ana Maria Corraliza, Victòria Gudiño, Alba Garrido-Trigo, Ángela Sanzo-Machuca, Marc Buendia, Miriam Esteller, Lisseth Robbins, Maria Teresa Rodrigo‐Calvo, Maria Carme Masamunt, Angel Giner, Marta Gallego, Íngrid Ordás, Agnes Fernandez-Clotet, , Azucena Salas

The paper investigates resistance to JAK inhibitors in UC by analyzing single-cell RNA data from responders and non-responders, identifying hyperactivation of myeloid cells as a key mechanism.

Journal of Crohn s and Colitis · May 5, 2025Peer-reviewedImmunology

Mitochondrial dysfunction: unraveling the elusive biology behind anti-TNF response during ulcerative colitis

Dimitrios Kioroglou, Ainize Peña‐Cearra, Ana Maria Corraliza, Iratxe Seoane, Janire Castelo, Julián Panés, Laura Gómez-Irwin, Iago Rodríguez–Lago, Jone Ortiz de Zárate, Miguel Fuertes, Itziar Martín‐Ruiz, Monika González-Lopez, Ana María Aransay, Azucena Salas, Héctor Rodrı́guez, , Urko M. Marigorta

The paper investigates the relationship between mitochondrial dysfunction and anti-TNF treatment response in ulcerative colitis using both murine and human samples, finding metabolic pathways and mitochondrial homeostasis are involved.

Inflammatory Bowel Diseases · Feb 13, 2025Peer-reviewedImmunology