Author

Michael Milyavsky

Broad InstituteORCID 0000-0002-5083-0809Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:experimental assay· 1 Research Focus:fine-mapping· 1 Research Focus:gene regulatory network inference· 1 Research Focus:GWAS / association mapping· 1 Assay:GWAS summary statistics· 1 Research Focus:host-pathogen & immune response· 1 Approach:statistical model· 1

Frequent Co-Authors

Aarno V. PalotieiD· 1 shared Aitzkoa Lopez de Lapuente PortillaiD· 1 shared Aki Samuli HavulinnaiD· 1 shared Alexander G. BickiD· 1 shared Amilia MeiriD· 1 shared Aviv RegeviD· 1 shared Benjamin M. NealeiD· 1 shared Björn NilssoniD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperGenetics

Aarno V. PalotieORCID

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2 papers

Recent·Most cited

Sustained exposure to CAR-T cell secretome impairs human Hematopoietic Stem Cell function and is reversible by dual TNFα-IFNγ blockade

Siva Sai Naga Anurag Muddineni, Diana Rasoulouniriana, Amilia Meir, Danielle Geller, Debanjan Singha Roy, Einat Tako, Neta Solomon, Tal Avraham, Yael Raz, Rony Chen, Eric Shifrut, Elad Jacoby, Michael Milyavsky

This study shows that sustained exposure of human HSPCs to CAR-T cell secretome impairs their function, but this effect is reversible with dual TNFα-IFNγ blockade.

bioRxiv · Mar 20, 2026 · PreprintCell Biology

Genetic predisposition to myeloproliferative neoplasms implicates hematopoietic stem cell biology

Erik L. Bao, Satish Kumar Nandakumar, Xiaotian Liao, Alexander G. Bick, Juha M Karjalainen, Marcin Tabaka, Olga I. Gan, Aki Samuli Havulinna, Tuomo Kiiskinen, Caleb A. Lareau, Aitzkoa Lopez de Lapuente Portilla, Bo Li, Connor A. Emdin, Veryan Codd, Christopher P. Nelson, , Vijay G. Sankaran

This study identifies 14 genome-wide significant loci and a polygenic signature for myeloproliferative neoplasms, implicating hematopoietic stem cell function and self-renewal in disease risk.

bioRxiv · Oct 8, 2019 · PreprintGenetics