Author

Oliva C. Fernández Cimadevilla

Hospital Universitario San AgustínORCID 0000-0002-5163-3882Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

Álvaro F. FernándeziD· 2 shared Gemma G. Martínez-García· 2 shared Guillermo MariñoiD· 2 shared Isaac Tamargo-GómeziD· 2 shared Alejandro P. UgaldeiD· 1 shared Andrés Corrochano-RuiziD· 1 shared Aurora AstudilloiD· 1 shared Carlos López-Otı́niD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersPathology

Álvaro F. FernándezORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

PathologybioRxivPreprintSep 29, 2025

ATG4D loss leads to late-onset cardiomyopathy and stress-induced heart failure in mice, and its repression marks maladaptive cardiac remodeling in humans

Guillermo Mariño, Isaac Tamargo-Gómez, Raquel García-López…·Álvaro F. Fernández

This study shows that ATG4D deficiency in mice causes late-onset cardiomyopathy and stress-induced heart failure, and ATG4D repression is linked to human cardiac remodeling.

DOI 10.1101/2025.09.29.678527

mousehuman

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Cell BiologybioRxivPreprintJan 25, 2025

Combined loss of ATG4B, ATG4C and ATG4D impairs systemic autophagy and triggers accelerated aging in mice

Gemma G. Martínez-García, María Fernanda Suárez, Mar Rodríguez-Santamaría…·Guillermo Mariño

This study shows that loss of ATG4B, ATG4C, and ATG4D in mice causes systemic autophagy defects, leading to accelerated aging and shortened lifespan.

1 citation

mouseexperimental assay

𝕏 PostBluesky