Author

Masahiro Fujimuro

Kyoto Pharmaceutical UniversityORCID 0000-0002-5165-0789Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

Akira SawaiD· 1 shared Atsushi KamiyaiD· 1 shared Atsushi SaitoiD· 1 shared Edwin C. T. OhiD· 1 shared Gary S. HaywardiD· 1 shared Gerd G. Maul· 1 shared Hannah Murdoch· 1 shared Ho NamkungiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperDevelopmental Biology

Akira SawaORCID

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2 papers

Recent·Most cited

Characterization of the Kaposi's sarcoma-associated herpesvirus terminase complex component ORF29

Yuki Iwaisako, Karin Shimizu, Youichi Suzuki, Takashi Nakano, Masahiro Fujimuro

This study identifies ORF29 as a critical component of the KSHV terminase complex, essential for capsid formation and TR cleavage, and shows it interacts with itself and ORF7.

Microbiology Spectrum · Sep 9, 2025Peer-reviewedMicrobiology

DISC1-PML protein interaction for congenital CMV infection-induced cortical neural progenitor deficit: perturbance of host signaling via viral IE1

Atsushi Saito, Stephanie K. Tankou, Kazuhiro Ishii, Makiko Sakao‐Suzuki, Edwin C. T. Oh, Hannah Murdoch, Ho Namkung, Sunday Adelakun, Keiko Furukori, Masahiro Fujimuro, Paolo Salomoni, Gerd G. Maul, Gary S. Hayward, Qiyi Tang, Robert H. Yolken, , Akira Sawa

This study identifies the role of CMV viral protein IE1 in disrupting host DISC1-PML interaction, leading to neural progenitor deficits in congenital CMV infection.

bioRxiv · Sep 3, 2025 · PreprintDevelopmental Biology