Author

Mieko Morishima

Doshisha UniversityORCID 0000-0002-5178-7531Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Approach:simulation· 1 Assay:spatial transcriptomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Ayana Haratake· 1 shared Ayumi Hirano‐IwataiD· 1 shared Hideaki YamamotoiD· 1 shared Jun TakahashiiD· 1 shared Kaneyasu NishimuraiD· 1 shared Kazuto KobayashiiD· 1 shared Kazuyuki TakataiD· 1 shared Kyohei FujitaiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperNeuroscience

Ayana Haratake

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Thalamocortical credit routing from the basal ganglia and cerebellum: anatomical constraints and circuit principles

Sho Tanno, Mieko Morishima, Kyohei Fujita, Shigeki Kato, Kazuto Kobayashi, Yasuo Kawaguchi, Yoshikazu Isomura, Riichiro Hira

The paper investigates how thalamocortical pathways from the basal ganglia and cerebellum route credit signals to cortical circuits, revealing anatomical segregation and plasticity mechanisms that enhance learning.

bioRxiv · Oct 8, 2026 · PreprintNeuroscience

Modeling interregional propagation of α-synuclein in human striatal-midbrain assembloids

Kaneyasu Nishimura, Naoya Amimoto, Yuya Sato, Mieko Morishima, Mamoru Sakaibara, Ayana Haratake, Mai Nakano, Naoko Kaneko, Hideaki Yamamoto, Ayumi Hirano‐Iwata, Takashi Tanii, Jun Takahashi, Kazuyuki Takata, Yoshito Masamizu

This study uses human striatal-midbrain assembloids to show that α-synuclein pathology spreads from the striatum to midbrain dopaminergic neurons, causing functional impairment.

bioRxiv · May 11, 2026 · PreprintNeuroscience