Marta Magnowska

The paper investigates stress resilience in mice, showing it involves structural, functional, and molecular changes in synapses, particularly through palmitoylation of synaptic proteins and dendritic spine remodeling.

A mutation in the TRAP1 gene causes autism-like behaviors in male mice, suggesting a role for mitochondrial protein homeostasis in male-specific autism.

This study identifies plasmin-mediated cleavage of EphA4 at central amygdala inhibitory synapses as a mechanism controlling anxiety through modulation of GABA-receptor anchoring and dendritic spine morphology.

This paper introduces an automatic method for dendritic spine shape taxonomy and behavior modeling, finding that learning effects are linked to ensemble changes rather than individual shape classes.