Author

Kristina Dietert

Freie Universität BerlinORCID 0000-0002-5667-6750Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Organism:human· 1

Frequent Co-Authors

Achim Dieter GruberiD· 2 shared Martin WitzenrathiD· 2 shared Alexander Taylor· 1 shared Andreas Christian HockeiD· 1 shared Andreas Gebhardt· 1 shared Anne-Katrin Bothe· 1 shared Birgitt Gutbier· 1 shared Capnetz Study Group· 1 shared

Activity by Year

2024-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersImmunology

Achim Dieter GruberORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Neutrophil-chemoattractant CXCL5 induces lung barrier permeability in acute lung injury

Sarah Berger, Cengiz Goekeri, Peter Pennitz, Birgitt Gutbier, Laura Michalick, Karen Hoffmann, Elena López-Rodríguez, Vladimir Gluhovic, Sandra-Maria Wienhold, Ulrike Behrendt, Alexander Taylor, Kristina Dietert, Holger Kirsten, Sandra Kunder, Kristina Mueller, , Geraldine Nouailles

This study identifies CXCL5 as a factor that increases alveolar-epithelial barrier permeability in acute lung injury, independent of neutrophil recruitment.

Mucosal Immunology · Oct 11, 2025Peer-reviewedImmunology

Expression of human CEACAMs promotes inflammation and organ damage during systemic Candida albicans infection in mice

Esther Klaile, Mario M. Müller, Johannes Sonnberger, Anne-Katrin Bothe, Saskia Brehme, Juliet Ehrenpfordt, Tilman E. Klassert, Sabina Kuhn, Kristina Dietert, Olivia Kershaw, Jan‐Philipp Praetorius, Marc Thilo Figge, Torsten Thomas Bauer, Andreas Gebhardt, Gita Mall, , Hortense Slevogt

This study shows that human CEACAM receptors in mice worsen inflammation and organ damage during Candida albicans infection, leading to shorter survival.

bioRxiv · Mar 29, 2025 · PreprintImmunology

IGF1R is protective in pneumococcal pneumonia

Matthias Felten, Luiz Gustavo Teixeira Alves, Eleftheria Letsiou, Elvira Alfaro‐Arnedo, Sergio Piñeiro‐Hermida, Icíar P. López, Kristina Dietert, Theresa Firsching, Achim Dieter Gruber, Diana Fatykhova, Andreas Christian Hocke, Tilman Lingscheid, Jasmin Lienau, Gernot G. U. Rohde, Capnetz Study Group, , Martin Witzenrath

This study shows that IGF1R deficiency worsens pneumococcal pneumonia in mice, with increased lung damage and bacterial load, suggesting IGF1R has a protective role.

bioRxiv · Sep 11, 2024 · PreprintMolecular Biology