Author

Capnetz Study group

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Gernot G. U. RohdeiD· 2 shared A. Diefenbach· 1 shared A. Fagundes· 1 shared A. Rabes· 1 shared Abhishek Kumar SinghiD· 1 shared Achim Dieter GruberiD· 1 shared Andreas Christian HockeiD· 1 shared Anne K Koch· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersMicrobiology

Gernot G. U. RohdeORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

HERC4 limits oxidative stress-induced DNA damage during bacterial and viral-bacterial infection

Clemens Cammann, Vanessa Gering, Thomas Sura, Abhishek Kumar Singh, Julia Désirée Boehme, Eylin Topfstedt, Anne K Koch, Ulrike Ritter, Karsten Becker, Dunja Bruder, Ulrike Blohm, Hortense Slevogt, Sandra Maaß, Gernot G. U. Rohde, Jan Rupp, , Ulrike Seifert

The paper identifies HERC4 as a regulator of ROS-induced DNA damage during bacterial and viral-bacterial infections, showing its role in DNA repair and potential as a therapeutic target.

bioRxiv · Jul 4, 2026 · PreprintMicrobiology

IGF1R is protective in pneumococcal pneumonia

Matthias Felten, Luiz Gustavo Teixeira Alves, Eleftheria Letsiou, Elvira Alfaro‐Arnedo, Sergio Piñeiro‐Hermida, Icíar P. López, Kristina Dietert, Theresa Firsching, Achim Dieter Gruber, Diana Fatykhova, Andreas Christian Hocke, Tilman Lingscheid, Jasmin Lienau, Gernot G. U. Rohde, Capnetz Study Group, , Martin Witzenrath

This study shows that IGF1R deficiency worsens pneumococcal pneumonia in mice, with increased lung damage and bacterial load, suggesting IGF1R has a protective role.

bioRxiv · Sep 11, 2024 · PreprintMolecular Biology

IL-33 controls IL-22-dependent antibacterial defense by modulating the microbiota

I. Roewekamp, L. Maschirow, A. Rabes, F. Fiocca Vernengo, G. A. Heinz, M.-F. Mashreghi, S. Caesar, M. Milek, A. Fagundes, S.-M. Wienhold, G. Nouailles, L. Yao, D. Bruder, J. Boehme, M. Puzianowska-Kuznicka, , B. Opitz

This study shows IL-33 reduces IL-22 production in innate lymphoid cells, increasing resistance to Streptococcus pneumoniae infection, and links genetic variation in IL-33 signaling to human pneumonia.

bioRxiv · Jul 19, 2023Immunology