Author

Polina M. Krassovskaia

Atrium Health Wake Forest BaptistORCID 0000-0002-6271-3036Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Molecular Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:ATAC-seq· 2 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Brett R. ChrestiD· 2 shared James T. HageniD· 2 shared Joseph M. McClungiD· 2 shared Kelsey H. Fisher‐WellmaniD· 2 shared McLane M. MontgomeryiD· 2 shared Raphael Taiwo ArulebaiD· 2 shared Thomas D. GreeniD· 2 shared Aaron David SchimmeriD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPhysiology

Brett R. ChrestORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

PhysiologyScience AdvancesPeer-reviewedJul 10, 2026

The MEK inhibitor trametinib incurs mitochondrial injury and induces innate immune responses in the mouse heart

Kelsey H. Fisher‐Wellman, Richard D. Lutze, Logan G. Kirkland…·Brian C. Jensen

This study shows trametinib causes mitochondrial injury and innate immune activation in mouse hearts, leading to cardiomyopathy and heart failure.

DOI 10.1126/sciadv.aeb2695

𝕏 PostBluesky

Molecular BiologybioRxivPreprintFeb 28, 2026

A full-length single nuclei transcriptomic atlas of human skeletal muscle insulin resistance

Katie L. Whytock, Adeline Divoux, James Vazquez…·Bret H. Goodpaster

The paper generates a nucleus-resolved transcriptomic atlas of skeletal muscle insulin resistance using single-nucleus transcriptomics and stable isotope-labeled clamps, identifying cell-specific molecular programs and targets.

DOI 10.64898/2026.02.26.708250

𝕏 PostBluesky

Cancer BiologyScience AdvancesPeer-reviewedApr 9, 2025

Acute myeloid leukemia mitochondria hydrolyze ATP to resist chemotherapy

James T. Hagen, McLane M. Montgomery, Raphael Taiwo Aruleba…·Kelsey H. Fisher‐Wellman

This study identifies a bioenergetic mechanism in AML cells where mitochondria consume ATP to resist chemotherapy, offering a target for improving treatment.

30 citationstop 2%

𝕏 PostBluesky