Author

Thomas D. Green

Atrium Health Wake Forest BaptistORCID 0009-0008-5435-6957Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Physiology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:ATAC-seq· 2 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Joseph M. McClungiD· 3 shared Kelsey H. Fisher‐WellmaniD· 3 shared Brett R. ChrestiD· 2 shared James T. HageniD· 2 shared McLane M. MontgomeryiD· 2 shared Polina M. KrassovskaiaiD· 2 shared Raphael Taiwo ArulebaiD· 2 shared Tonya N. ZeczyckiiD· 2 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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3 shared papersPhysiology

Joseph M. McClungORCID

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3 papers

Recent·Most cited

PhysiologyScience AdvancesPeer-reviewedJul 10, 2026

The MEK inhibitor trametinib incurs mitochondrial injury and induces innate immune responses in the mouse heart

Kelsey H. Fisher‐Wellman, Richard D. Lutze, Logan G. Kirkland…·Brian C. Jensen

This study shows trametinib causes mitochondrial injury and innate immune activation in mouse hearts, leading to cardiomyopathy and heart failure.

DOI 10.1126/sciadv.aeb2695

𝕏 PostBluesky

PhysiologyJournal of Vascular ResearchPeer-reviewedMay 9, 2026

Deletion of Murine Endothelial Bag3 Alters the Cellular Proteome Among Organs of Origin

Zoe S Terwilliger, Feifei Li, Ananya Pentakota…·Joseph M. McClung

This study shows that deleting BAG3 in endothelial cells uniquely alters the proteome of specific organs, with the most significant changes in skeletal muscle.

DOI 10.1159/000552442

mouseproteomics

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Cancer BiologyScience AdvancesPeer-reviewedApr 9, 2025

Acute myeloid leukemia mitochondria hydrolyze ATP to resist chemotherapy

James T. Hagen, McLane M. Montgomery, Raphael Taiwo Aruleba…·Kelsey H. Fisher‐Wellman

This study identifies a bioenergetic mechanism in AML cells where mitochondria consume ATP to resist chemotherapy, offering a target for improving treatment.

30 citationstop 2%

𝕏 PostBluesky