Barbara A. Bensing

This study shows that sialic acid-binding bacterial lectins enhance HIV-1 infectivity in vitro and mucosal transmission in humanized mice by promoting virus-cell attachment and spread.

This study shows that sialic acid identity influences, but does not solely determine, host tropism of sialoglycan-binding streptococci by testing binding preferences of SLBRs to Neu5Ac and Neu5Gc.

The paper develops sialoglycan-recognizing probes (SGRPs) to systematically study mammalian sialoglycome dynamics using established laboratory techniques.