Author

Shabir Zargar

National Cancer InstituteORCID 0000-0002-6384-6359Unclaimed ORCID profile · follow to get notified of new preprints

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Benjamin Z. StantoniD· 2 shared Frederic G. BarriD· 2 shared Pawan Kumar RautiD· 2 shared Rachel A. HoffmaniD· 2 shared Bishwanath ChatterjeeiD· 1 shared Hana KimiD· 1 shared Hana KimiD· 1 shared Puspa Raj PandeyiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCancer Biology

Benjamin Z. StantonORCID

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2 papers

Recent·Most cited

Cancer BiologyMolecular Cancer ResearchPeer-reviewedAug 17, 2026

MIR17HG Expression Is Transcriptionally Regulated by PAX3::FOXO1 and MYCN 1 and is Necessary for Oncogenic Activity in Fusion-Positive Rhabdomyosarcoma

Shabir Zargar, Pawan Kumar Raut, Hana Kim…·Frederic G. Barr

This study identifies PAX3::FOXO1 and MYCN as cooperative regulators of MIR17HG expression in fusion-positive rhabdomyosarcoma and shows miR-17-92 is necessary for oncogenic activity.

DOI 10.1158/1541-7786.mcr-26-0074

humangenetic perturbation / CRISPR

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Cancer BiologybioRxivPreprintDec 2, 2025

Role of Myc family proteins in transcriptional regulation of growth and oncogenic transformation in fusion-positive rhabdomyosarcoma

Bishwanath Chatterjee, Pawan Kumar Raut, Hana Kim…·Frederic G. Barr

This study investigates the role of Myc family proteins in FP-RMS, showing MYCN or MYC dominance in some cases and functional redundancy in others, with both contributing to oncogenic properties.

DOI 10.64898/2025.12.01.690506

humanbulk RNA-seq

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