Author

Catherine K. Yeung

University of WashingtonORCID 0000-0002-6556-6641Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Assay:proteomics· 1

Frequent Co-Authors

Edward J. KellyiD· 2 shared James W. MacDonaldiD· 2 shared Jonathan HimmelfarbiD· 2 shared Theo K. BammleriD· 2 shared Angela R. Zheng· 1 shared Anish Mahadeo· 1 shared Eric W. HsuiD· 1 shared Kayenat S Aryeh· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersPharmacology & Toxicology

Edward J. KellyORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Voclosporin Preserves Mitochondrial Function Compared With Cyclosporine A in Perfused Human Proximal Tubule Microphysiological Systems

Kayenat S Aryeh, Yik Pui Tsang, Eric W. Hsu, Catherine K. Yeung, James W. MacDonald, Theo K. Bammler, Jonathan Himmelfarb, Linda M. Rehaume, Edward J. Kelly

This study compares voclosporin and cyclosporine A in 3D kidney MPS, finding that voclosporin preserves mitochondrial function and reduces proteostatic stress compared to cyclosporine A.

bioRxiv · Jul 11, 2026 · PreprintPharmacology & Toxicology

Evaluation of toxicological mechanisms of ochratoxin-A in human primary proximal tubule epithelial cells

Anish Mahadeo, Theo K. Bammler, James W. MacDonald, Angela R. Zheng, Catherine K. Yeung, Jonathan Himmelfarb, Edward J. Kelly

This study investigates OTA toxicity in human kidney cells, showing it causes mitochondrial dysfunction, oxidative stress, and reduced energy production, contributing to kidney disease.

bioRxiv · Aug 29, 2025 · PreprintPharmacology & Toxicology