Author

Beata Wójciak‐Stothard

Imperial College LondonORCID 0000-0002-6607-7372Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

Adam FellowsiD· 1 shared Alexander J. AinscoughiD· 1 shared Allan LawrieiD· 1 shared Chien-Nien Chen· 1 shared Clare M. LloydiD· 1 shared Daria S. KostyuninaiD· 1 shared Hannah MaudeiD· 1 shared Harry J. WhitwelliD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

Adam FellowsORCID

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2 papers

Recent·Most cited

KLF6 in Pulmonary Hypertension: The Dual Role of Friend and Foe

Rehab Salah Saleh Alharbi, Merve Keleş, Nadia J. Fernandes, Hannah Maude, Adam Fellows, Richard Dafydd Williams, Chien-Nien Chen, Nathalie Lambie, Nik Matthews, May Al‐Sahaf, Sam N. Barnett, Minzhe Guo, Lan Zhao, Allan Lawrie, Jeffrey A. Whitsett, , Beata Wójciak‐Stothard

This study identifies KLF6 as a key driver of endothelial dysfunction in pulmonary arterial hypertension, showing its role in promoting vascular remodelling and angioproliferative lesions.

Communications Biology · Jun 15, 2026Peer-reviewedCell Biology

Organ on chip model of respiratory vascular interactions under COPD relevant oxidative stress

Maike Haensel, Rosie Millns, Harry J. Whitwell, Alexander J. Ainscough, Joseph M. Sherwood, Lucas Breuil, Daria S. Kostyunina, Clare M. Lloyd, Beata Wójciak‐Stothard

This study develops an organ-on-chip model to investigate respiratory-vascular interactions under oxidative stress, revealing how airway injury drives vascular inflammation and remodeling in COPD.

bioRxiv · Jun 9, 2026 · PreprintBioengineering