Author

Hanjun Li

Shanghai Jiao Tong UniversityORCID 0000-0002-7017-8782Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Hanjun Li1 paper · Shanghai Cancer Institute, Ren Ji Hospital, Shanghai Jiao Tong University School of Medicine;

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Assay:metabolomics· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

Cegui HuiD· 1 shared Chenwei Yuan· 1 shared H Y Wang· 1 shared Jinsong LuiD· 1 shared Jun GuiiD· 1 shared Junqi Cui· 1 shared Kai YuaniD· 1 shared Li PengiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

Cegui HuORCID

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2 papers

Recent·Most cited

Piezo1 transduces mechanical signals to inhibit osteoclast fusion and coordinate bone homeostasis

Ying Wang, H Y Wang, Tianyou Kan, Junqi Cui, Xuran Li, Kai Yuan, Liao Wang, Mengning Yan, Linyang Chu, Hanjun Li, Z-F Yu

This study shows that Piezo1 transduces mechanical signals to inhibit osteoclast fusion and maintain bone homeostasis by activating a signaling pathway that suppresses osteoclast differentiation.

bioRxiv · Jun 6, 2026 · PreprintCell Biology

Cancer-associated adipocytes mediate CD8+T cell dysfunction via FGF21-driven lipolysis

Sumiya Dalangood, Cegui Hu, Chenwei Yuan, Xiang Olivia Li, Wen Qiao, Hanjun Li, Rongyu Zhang, Luying Li, Li Peng, Xiang Yu, Wenjin Yin, Jinsong Lu, Jun Gui

This study identifies FGF21-driven lipolysis in cancer-associated adipocytes as a mechanism causing CD8+T cell dysfunction, showing that targeting this process can enhance cancer immunotherapy.

Cell Reports · Nov 1, 2025Peer-reviewedCancer Biology