Author

Yu‐shin Sou

Juntendo UniversityORCID 0000-0002-7219-9106Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:differential expression· 1 Assay:proteomics· 1

Frequent Co-Authors

Masaaki KomatsuiD· 2 shared Satoko Komatsu‐Hirota· 2 shared Satoshi WaguriiD· 2 shared Elena RyzhiiiD· 1 shared Gaoxin MaoiD· 1 shared Hideaki MorishitaiD· 1 shared Hikaru TsuchiyaiD· 1 shared Manabu AbeiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCell Biology

Masaaki KomatsuORCID

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2 papers

Recent·Most cited

Cell BiologyEMBO ReportsPeer-reviewedMay 28, 2025

KEAP1 retention in phase-separated p62 bodies drives liver damage in autophagy-deficient conditions

Shuhei Takada, Nozomi Shinomiya, Gaoxin Mao…·Masaaki Komatsu

The paper shows that KEAP1 retention in p62 bodies and NRF2 activation cause liver damage when autophagy is impaired, and that blocking p62-KEAP1 interactions reduces this damage.

12 citations

mouseproteomics

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Cell BiologybioRxivPreprintMay 29, 2024

Loss of SPNS1, a lysosomal transporter, in the nervous system causes dysmyelination and white matter dysplasia

Yoshinobu Ichimura, Yuki Sugiura, Yoshinori Katsuragi…·Masaaki Komatsu

This study shows that loss of SPNS1 in the nervous system causes dysmyelination and white matter dysplasia in mice, suggesting a role for SPNS1 mutations in human congenital white matter disorders.

1 citation

mouseexperimental assay

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