Author

Alessandro Gonzalez Salerno

University of MiamiORCID 0000-0002-7688-5904Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Assay:proteomics· 1

Frequent Co-Authors

Amarylis Claudine Bonito Azeredo WanscheliD· 2 shared Andrew Victor SchallyiD· 1 shared Angeliki DaiouiD· 1 shared Antigone LazouiD· 1 shared Christine KottaridiiD· 1 shared Czarina P. Sobejana· 1 shared Daniel A. RodrigueziD· 1 shared Derek Michael DykxhoorniD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersPhysiology

Amarylis Claudine Bonito Azeredo WanschelORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Estrogen-Nitric Oxide Signaling Modulates Mitochondrial Dynamics and Endothelial Lipid Handling to Protect Against Early Atherosclerosis

Emmalie R Spry, Hannah Strcula, Giovana Abe Mascoli, Czarina P. Sobejana, Mirabella Zingales, Marta Helena Krieger, Alessandro Gonzalez Salerno, Amarylis Claudine Bonito Azeredo Wanschel

This study shows estrogen and nitric oxide signaling reduce oxLDL uptake and oxidative stress in endothelial cells, modulate mitochondrial dynamics, and decrease inflammation, protecting against early atherosclerosis.

bioRxiv · May 15, 2026 · PreprintPhysiology

Oxygen-independent, hormonal control of HIF-1α regulates the developmental and regenerative growth of cardiomyocytes.

Amarylis Claudine Bonito Azeredo Wanschel, Angeliki Daiou, Jeffim N. Kuznetsoff, Stefan Kurtenbach, Katerina Petalidou, Thomai Mouskeftara, Helen Gika, Daniel A. Rodriguez, Eleftherios I. Papadopoulos, Georgios Siokatas, Polyxeni P. Sarri, Kyriaki Karava, Efthimios Tsivoglou, Christine Kottaridi, Alessandro Gonzalez Salerno, , Joshua M. Hare

This paper identifies an O2-independent HIF-1 mechanism regulated by GH/IGF1/SST signaling that controls cardiomyocyte growth through metabolic shifts.

bioRxiv · Jan 31, 2022 · PreprintDevelopmental Biology