Author

David T. Paik

Stanford Cardiovascular InstituteORCID 0000-0002-7830-312XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Genetic & Genomic Medicine· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 3 Research Focus:differential expression· 2 Organism:human· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:fine-mapping· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Joseph C. WuiD· 3 shared Aabida SaferaliiD· 1 shared Adithya BalasubramanianiD· 1 shared Albert V. SmithiD· 1 shared Alex P. ReineriD· 1 shared Alison Schroer Vander RoestiD· 1 shared Amber Beitelshees· 1 shared Angel C. Y. MakiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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3 shared papersGenetic & Genomic Medicine

Joseph C. WuORCID

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3 papers

Recent·Most cited

Genetic & Genomic MedicineSciencePeer-reviewedJul 16, 2026

Cross-cohort analysis of expression and splicing quantitative trait loci in TOPMed

Peter Orchard, Thomas W. Blackwell, Linda Kachuri…·Jeffrey L. Curtis

The paper analyzes expression and splicing quantitative trait loci (e/sQTL) across multiple tissues in the TOPMed dataset and finds that secondary e/sQTL signals are still discoverable, with many GWAS signals colocalizing with these e/sQTL.

16 citationstop 1%

humanbulk RNA-seq

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Molecular BiologybioRxivPreprintJun 8, 2023

Multi-scale models reveal hypertrophic cardiomyopathy MYH7 G256E mutation drives hypercontractility and elevated mitochondrial respiration

Soah Lee, Alison Schroer Vander Roest, Cheavar A. Blair…·Daniel Bernstein

This study investigates the effects of the MYH7 G256E mutation on myosin function across multiple scales, finding increased hypercontractility and mitochondrial respiration.

2 citations

humanscRNA-seq

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Developmental BiologyEuropean Heart JournalPeer-reviewedMay 16, 2021

Endocardial/endothelial angiocrines regulate cardiomyocyte development and maturation and induce features of ventricular non-compaction

Siyeon Rhee, David T. Paik, Johnson Y. Yang…·Kristy Red‐Horse

This study identifies angiocrine factors secreted by endocardial and endothelial cells that regulate cardiomyocyte development and maturation, contributing to ventricular non-compaction in Ino80-deficient mouse hearts.

72 citationstop 4%

mousescRNA-seq

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