Author

Jonathan Dias

Centre Hospitalier de l’Université de MontréalORCID 0000-0002-7842-0115Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Immunology· 4 Microbiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 5 Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 3 Approach:statistical model· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:flow cytometry· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

Petronela AncuțaiD· 5 shared Jean‐Pierre RoutyiD· 4 shared Soumia Khalfi· 3 shared Tomas Raul Wiche SalinasiD· 3 shared Abdelali Filali-MouhimiD· 2 shared Andrés FinziiD· 2 shared Carl Chartrand‐LefebvreiD· 2 shared Cecile L. TremblayiD· 2 shared

Activity by Year

2025-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

5 shared papersImmunology

Petronela AncuțaORCID

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

ImmunologybioRxivPreprintAug 25, 2026

ALDH Activity in Monocytes is Associated with Subclinical Coronary Atherosclerosis in Treated People with HIV-1

Jonathan Dias, Mohamed El Far, Kevin E. Boczar…·Petronela Ancuța

The paper links ALDH activity in monocytes to subclinical coronary atherosclerosis in HIV-1 treated individuals, independent of traditional risk factors.

DOI 10.64898/2026.08.20.746037

humanflow cytometry

𝕏 PostBluesky

ImmunologybioRxivPreprintAug 24, 2026

REV-ERBα/β Targeting Transcriptionally Reprograms HIV-Infected CD4+ T-Cells for Increased Viral Reactivation but Limited Virion Spread

Christ Dominique Ngassaki Yoka, Jean-Philippe Goulet, Soumia Khalfi…·Petronela Ancuța

This study shows that inhibiting REV-ERBβ increases HIV-1 reactivation in CD4+ T-cells but limits virion spread.

DOI 10.64898/2026.08.20.746039

humanbulk RNA-seq

𝕏 PostBluesky

MicrobiologybioRxivPreprintMay 4, 2026

Identification of the cellular transcription factor KLF16 as a novel repressive epigenetic repressor of HIV-1 transcription

Marion Santangelo, Maryam Bendoumou, Antoine Dutilleul…·Carine M. Van Lint

This study identifies KLF16 as a repressive transcription factor that inhibits HIV-1 transcription by competing with Sp1 and recruiting repressive complexes.

DOI 10.64898/2026.05.02.722432

humanChIP-seq

𝕏 PostBluesky

ImmunologyCellsPeer-reviewedNov 4, 2025

Alterations in Circulating T-Cell Subsets with Gut-Homing/Residency Phenotypes Predict HIV-1 Status and Subclinical Atherosclerosis

Etiene Moreira Gabriel, Jonathan Dias, Abdelali Filali-Mouhim…·Petronela Ancuța

The paper identifies specific T-cell subsets with gut-homing/residency markers that predict HIV status and subclinical atherosclerosis in ART-treated individuals.

1 citation

humanflow cytometry

𝕏 PostBluesky

ImmunologybioRxivPreprintJan 31, 2025

Humanized Bone Marrow-Liver-Thymus Mice for Studying HIV-1 Persistence in Liver and Lung CD4+ T and Myeloid Cell Subsets during Antiretroviral Therapy

Amélie Cattin, Tram N. Q. Pham, Jonathan Dias…·Petronela Ancuța

This study uses humanized mice to compare HIV-1 persistence in liver/lung macrophages and CD4+ T-cells during ART, finding macrophages contribute less to viral reservoirs than T-cells.

1 citation

humanexperimental assay

𝕏 PostBluesky