Petronela Ancuța

The paper links ALDH activity in monocytes to subclinical coronary atherosclerosis in HIV-1 treated individuals, independent of traditional risk factors.

This study shows that inhibiting REV-ERBβ increases HIV-1 reactivation in CD4+ T-cells but limits virion spread.

This study identifies KLF16 as a repressive transcription factor that inhibits HIV-1 transcription by competing with Sp1 and recruiting repressive complexes.

The paper identifies specific T-cell subsets with gut-homing/residency markers that predict HIV status and subclinical atherosclerosis in ART-treated individuals.

This study compares SARS-CoV-2 immune responses in head and neck cancer patients and controls, finding similar antibody levels but higher convalescent/hybrid immunity and stable B-cell responses.

This study uses humanized mice to compare HIV-1 persistence in liver/lung macrophages and CD4+ T-cells during ART, finding macrophages contribute less to viral reservoirs than T-cells.

This study shows metformin reduces HIV-1 virion release and increases antibody recognition of infected CD4+ T-cells by modulating BST2 and Bcl-2 expression in HIV-positive individuals on ART.

This study shows retinoic acid enhances R5 HIV-1 replication in macrophages through CCR5/SAMHD1 and mTOR pathways.

This study identifies immunological markers associated with subclinical coronary artery atherosclerosis in HIV+ individuals on ART, finding reduced Th17/Treg ratios and increased non-classical monocyte frequencies correlate with atherosclerosis burden.