Author

Ana Vasconcelos‐Ferreira

Centro de Neurociências e Biologia CelularORCID 0000-0002-8968-0641Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Organism:human· 1

Frequent Co-Authors

Dina PereiraiD· 2 shared Luis Pereira de AlmeidaiD· 2 shared Ana Margarida SalgueiroiD· 1 shared Ana Rita Fernandes· 1 shared Carlos A. MatosiD· 1 shared Célia Alexandra AveleiraiD· 1 shared Cláudia CavadasiD· 1 shared Clévio NóbregaiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersNeuroscience

Dina PereiraORCID

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2 papers

Recent·Most cited

Nuclear aging in polyglutamine-induced neurodegeneration

Dina Pereira, Janete Cunha‐Santos, Ana Vasconcelos‐Ferreira, Joana Duarte‐Neves, Isabel Onofre, Célia Alexandra Aveleira, Vítor Carmona, Luísa Cortes, Sara M. Lopes, Diana Duarte Lobo, Inês M. Martins, Ana Margarida Salgueiro, Nélio Gonçalves, Cláudia Cavadas, Luis Pereira de Almeida

This study investigates the role of aging in Machado-Joseph disease by analyzing age-related markers in human and animal models, finding decreased lamins and ZMPSTE24 levels and nuclear abnormalities.

npj Aging · Aug 12, 2026Peer-reviewedNeuroscience

Gene Editing for ATXN3 Inactivation in Machado-Joseph disease: CRISPR-Cas9 as a Therapeutic Alternative to TALEN-Induced Toxicity

Sara Monteiro Lopes, Miguel Monteiro Lopes, Daniela Camargo de Oliveira, Magda M. Santana, Ana Rita Fernandes, Ana Vasconcelos‐Ferreira, Dina Pereira, Maria Casquinha, Clévio Nóbrega, Carlos A. Matos, Neville E. Sanjana, Patrick D. Hsu, F. Ann Ran, Lukasz J. Swiech, Le Cong, , Luís Pereira de Almeida

This study compares CRISPR-Cas9 and TALEN gene editing in a mouse model of Machado-Joseph disease, showing CRISPR-Cas9 reduces ATXN3 aggregates and delays disease progression.

bioRxiv · Feb 14, 2025 · PreprintNeuroscience