Author

Joakin Mori

Boston UniversityORCID 0000-0002-9033-8536Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Christopher M. HeaphyiD· 2 shared Alan Keith MeekeriD· 1 shared Anthony J. RizzoiD· 1 shared Carlo Lanza· 1 shared Christine Davis· 1 shared Ignaty LeshchineriD· 1 shared Jacqueline A. Brosnan‐CashmaniD· 1 shared Jiyoung KimiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCancer Biology

Christopher M. HeaphyORCID

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2 papers

Recent·Most cited

Functional inactivation of the telomerase chaperone TCAB1 primes cells for the activation of ALT in osteosarcoma

Joshua Keegan, Sydney Sorbello, Joakin Mori, Shugo Muratani, Ignaty Leshchiner, Christopher M. Heaphy, Rachel Litman Flynn

The paper identifies structural variations in TP53 that inactivate TCAB1, a telomerase chaperone, and show these variations are prevalent in ALT-positive osteosarcoma tumors.

bioRxiv · Jan 2, 2026 · PreprintCancer Biology

Loss of DAXX induces alternative lengthening of telomeres (ALT)-associated hallmarks in prostate cancer cells in a context-specific manner

Joakin Mori, Joseph Da, Jiyoung Kim, Anthony J. Rizzo, Christine Davis, Carlo Lanza, Jacqueline A. Brosnan‐Cashman, Alan Keith Meeker, Christopher M. Heaphy, Mindy Kim Graham

This study shows that DAXX mutations induce ALT-associated features in prostate cancer cells, but only in CWR22Rv1 cells, not in LAPC-4 cells.

bioRxiv · Aug 6, 2025 · PreprintCancer Biology