Author

Daniel Andrade Moreira

Fundação Oswaldo CruzORCID 0000-0002-9434-9582Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Assay:metagenomics· 1 Assay:proteomics· 1

Frequent Co-Authors

Athena Brianne Bradshaw· 1 shared Carolina FurtadoiD· 1 shared Chukwuma Great Udensi· 1 shared Daniel S. PeeperiD· 1 shared Danielle Gonçalves de Carvalho· 1 shared David T. PrideiD· 1 shared Emma Allen‐VercoeiD· 1 shared Heidi PaueriD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMicrobiology

Athena Brianne Bradshaw

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Enterocloster citroniae and related gut microbiome species modulate Vibrio cholerae biofilm formation through the production of bioactive small molecules

Heidi Pauer, Saeideh Nasiri, Nathalia Santos Magalhães, Vivian T. Nguyen, Nicole Victor Ferreira, Larissa D. Silva Ferreira, Athena Brianne Bradshaw, Katherine E. Kirby, Tyler Sabapathy, Chukwuma Great Udensi, Viktoriia Feofanova, Daniel Andrade Moreira, T. T. E. Parente, Jacob Wilde, David T. Pride, , L. Caetano M. Antunes

This study shows that Enterocloster citroniae and related gut microbes produce small molecules that inhibit Vibrio cholerae motility and promote biofilm formation, influencing its virulence.

Virulence · Sep 16, 2026Peer-reviewedMicrobiology

Resistance to BRAF inhibitors drives melanoma sensitivity to Chk1 inhibition

Danielle Gonçalves de Carvalho, Juliana C.N. Kenski, Daniel Andrade Moreira, Matheus Andrade Rajão, Oscar Krijgsman, Carolina Furtado, Mariana Boroni, João P. B. Viola, Daniel S. Peeper, Patrícia A. Possik

This study shows BRAF inhibitor-resistant melanoma cells are more sensitive to Chk1 inhibition due to increased replication stress, suggesting Chk1i could overcome MAPK inhibitor resistance.

Pharmacological Research · May 23, 2025Peer-reviewedCancer Biology