Marjan Rafat

This study shows that radiotherapy-induced vascular dysfunction leads to neutrophil activation and tumor cell colonization in mammary adipose tissue in lymphocyte-deficient mice.

The paper develops an agent-based model to show that unresolved inflammation after radiotherapy creates a tissue state that promotes tumor recurrence.

This paper develops a mammary-specific microfluidic device to study how radiotherapy affects vascular-immune interactions and promotes pre-metastatic niche formation.

This study shows that Th2-polarized CD4+ T-cells drive TNBC recurrence through TGFβ-induced pro-invasive signaling in irradiated mammary tissue.

The paper introduces nMATRIX, a material-cell platform that converts inflammatory cytokine signals into anti-inflammatory cues to reprogram macrophage phenotypes.

This study develops shelf-stable bioluminescence reagents and assay kits using capillary-assisted vitrification technology, demonstrating ambient storage and consistent performance in various assays.

This study shows that mammary fibroblasts release DAMPs via extracellular vesicles after ionizing radiation, with Rab27a regulating this process.