Author

A Beasley Lomazzi

Universidad de la República de UruguayORCID 0000-0002-9643-3163Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:electronic health records· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

AM FolleiD· 1 shared Ana Maria FerreiraiD· 1 shared Conny Leistner· 1 shared Federico CarrióniD· 1 shared Fernando ZamarreñoiD· 1 shared Jeremías Dutto· 1 shared Josep JulveiD· 1 shared Marcelo D. CostabeliD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperImmunology

AM FolleORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Echinococcus granulosus antigen B acts as an LPS-scavenging lipoprotein in vitro preventing TLR4-mediated activation of dendritic cells

Sofía Lagos Magallanes, A Beasley Lomazzi, Fernando Zamarreño, Federico Carrión, Martín Fló, Jeremías Dutto, Josep Julve, Marcelo D. Costabel, Mariana Maccioni, AM Folle, Ana Maria Ferreira

This study shows that Echinococcus granulosus antigen B binds LPS and prevents TLR4 activation in dendritic cells, inhibiting proinflammatory responses.

Infection and Immunity · Dec 16, 2025Peer-reviewedImmunology

The in-tissue molecular architecture of β-amyloid pathology in the mammalian brain.

Conny Leistner, Martin Wilkinson, Ailidh Burgess, Stanley Goodbody, Yong Xu, Susan A. Deuchars, Sheena E. Radford, Neil A. Ranson, René A. W. Frank

This study reveals the in-tissue molecular architecture of β-amyloid fibrils in a mouse model of Alzheimer's disease, showing their arrangement in lattices and their structural differences due to the Arctic mutation.

bioRxiv · Nov 8, 2022 · PreprintNeuroscience