Author

Ryan B. Jensen

Yale UniversityORCID 0000-0002-9844-0789Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Adeola A FagunloyeiD· 1 shared Arun Prakash MishraiD· 1 shared Connor P. McBrineiD· 1 shared Dylan Caylor· 1 shared Eileen A. Southon· 1 shared Fabricio S Justiniano· 1 shared Francesco Tomassoni‐ArdoriiD· 1 shared Gemma MooreiD· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperMolecular Biology

Adeola A FagunloyeORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

A single BRCA2 BRC repeat supports viability while multiple repeats ensure resilience under stress

Arun Prakash Mishra, Sounak Sahu, Satheesh Kumar Sengodan, Gemma Moore, Swati Priya, Natasha Oberoi, Julia R. Jensen, Eileen A. Southon, Melissa Galloux, Dylan Caylor, Teresa L. Sullivan, Sandra Sczerba Burkett, Mary E. Albaugh, Parirokh P. Awasthi, Francesco Tomassoni‐Ardori, , Shyam K. Sharan

This study shows that a single BRC repeat supports viability in mice but multiple repeats enhance resilience under oxidative stress.

bioRxiv · Jan 8, 2026 · PreprintGenetics

RAD51 Paralogs and RAD51 Paralog Complexes BCDX2 and CX3 Interact with BRCA2

Jacob G Thrasher, Adeola A Fagunloye, Fabricio S Justiniano, Kara Anne Bernstein, Ryan B. Jensen

This study identifies novel interactions between RAD51 paralogs and BRCA2, revealing a critical FxxA motif in RAD51B that mediates their binding.

bioRxiv · Oct 10, 2024 · PreprintMolecular Biology

Activation of Pol theta-mediated end joining begins during mitotic commitment

Connor P. McBrine, Ryan B. Jensen, Megan C. King

The paper identifies that TMEJ is optimally licensed during mitotic commitment and is regulated by PLK1, with DNA2 deficiency impairing TMEJ by delaying S-phase progression.

bioRxiv · Oct 3, 2024 · PreprintMolecular Biology