Author

Kristiano Ndoci

University of CologneORCID 0000-0002-9896-8196Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell-cell communication· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:spatial domain detection· 1

Frequent Co-Authors

Gulzar A. WaniiD· 3 shared Matteo BergamiiD· 3 shared Milica Jevtic· 3 shared Astrid C SchaussiD· 2 shared Elisa MotoriiD· 2 shared Felix GaedkeiD· 2 shared Laura Pérez-RevueltaiD· 2 shared Sandra M.V. KochaniD· 2 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

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3 shared papersNeuroscience

Gulzar A. WaniORCID

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Author profile →

3 papers

Recent·Most cited

Mitochondrial turnover at central GABAergic synapses governs vulnerability to epileptic seizures

Kristiano Ndoci, Abdulla W. Chihab, Kiruthika Ganesan, Milica Jevtic, Kelvin Maximilian Tofan, Gulzar A. Wani, Patric Pelzer, Jorge A. Soriano-Campos, Franka Odenthal, Vignesh Sakthivelu, Camilla Aurora Franchino, Laura Pérez-Revuelta, Jonas Benz, Anna‐Lena Schumacher, Felix Gaedke, , Matteo Bergami

This study shows that mitochondrial turnover at GABAergic synapses affects seizure vulnerability, with gene therapy reversing the effects.

bioRxiv · Jun 28, 2026 · PreprintNeuroscience

Enhanced mitochondrial fusion during a critical period of synaptic plasticity in adult-born neurons

Sandra M.V. Kochan, Meret Cepero Malo, Milica Jevtic, Hannah M. Jahn-Kelleter, Gulzar A. Wani, Kristiano Ndoci, Laura Pérez-Revuelta, Felix Gaedke, Iris Schäffner, Dieter Chichung Lie, Astrid C Schauss, Matteo Bergami

The paper shows that mitochondrial fusion during a critical period supports synaptic plasticity and circuit integration in adult-born neurons.

Neuron · Apr 5, 2024Peer-reviewedNeuroscience

Metabolic control of adult neural stem cell self-renewal by the mitochondrial protease YME1L

Gulzar A. Wani, Hans‐Georg Sprenger, Kristiano Ndoci, Srikanth Chandragiri, Richard Acton, Désirée Schatton, Sandra M.V. Kochan, Vignesh Sakthivelu, Milica Jevtic, Jens Michael Seeger, Stefan Müller, Patrick Giavalisco, Elena I. Rugarli, Elisa Motori, Thomas Langer, Matteo Bergami

This study identifies YME1L as a mitochondrial protease that regulates metabolic state transitions in adult neural stem cells, ensuring self-renewal and preventing premature differentiation.

Cell Reports · Feb 1, 2022Peer-reviewedNeuroscience