Author

Yomira Palacios

University of California Davis Medical CenterORCID 0000-0002-9968-2085Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Alain NdayisabaiD· 1 shared Alan P. BoyleiD· 1 shared Amy Krans· 1 shared Andrea CorteseiD· 1 shared Andrea Suárez· 1 shared Anke A. DijkstraiD· 1 shared Camille LoretiD· 1 shared Camille MummiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperNeuroscience

Alain NdayisabaORCID

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2 papers

Recent·Most cited

ZC4H2 loss of function is associated with temporal dysregulation of neural stem proliferation and neuron development

Yomira Palacios, Camille Loret, Viktoria Haghani, Kyle D. Fink, Julian A. N. M. Halmai

This study shows that ZC4H2 loss of function in human neural stem cells and organoids leads to premature neuronal differentiation and impaired neurogenesis, with potential therapeutic restoration through a codon-optimized transgene.

bioRxiv · Jan 15, 2026 · PreprintNeuroscience

AAGGG repeat expansions trigger RFC1-independent synaptic dysregulation in human CANVAS Neurons

Connor J. Maltby, Amy Krans, Samantha Grudzien, Yomira Palacios, Jessica Muiños, Andrea Suárez, Melissa J. Asher, Sydney Willey, Kinsey Van Deynze, Camille Mumm, Alan P. Boyle, Andrea Cortese, Alain Ndayisaba, Vikram Khurana, Sami J. Barmada, , Peter K. Todd

This study shows that AAGGG repeat expansions cause synaptic defects in CANVAS neurons independently of RFC1, with phenotypes corrected by repeat deletion.

Science Advances · Sep 4, 2024Peer-reviewedNeuroscience