Author

S. Sabater

Complejo Hospitalario Universitario de AlbaceteORCID 0000-0003-0111-9540Unclaimed ORCID profile · follow to get notified of new preprints

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S. Sabater1 paper

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Andrés Domínguez‐CalvoiD· 1 shared Borja BelandiaiD· 1 shared Cristina Garnés‐GarcíaiD· 1 shared Diego M. Fernández‐ArocaiD· 1 shared Francisco J. CimasiD· 1 shared Guillermo de CárceriD· 1 shared Ignacio Andrés· 1 shared Ignacio PalmeroiD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Andrés Domínguez‐CalvoORCID

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1 paper

Recent·Most cited

Cancer BiologyMolecular OncologyPeer-reviewedDec 3, 2024

The signaling axis CDK12-BRCA1 mediates dinaciclib associated radiosensitivity through p53-mediated cellular senescence.

Natalia García-Flores, Diego M. Fernández‐Aroca, Cristina Garnés‐García…·Ricardo Sánchez‐Prieto

This paper shows dinaciclib increases radiosensitivity in some cancer cell lines by inhibiting CDK12, reducing BRCA1, and promoting p53-dependent senescence.

7 citations

humanbulk RNA-seq

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