Author

Benjamin Devenney

Johns Hopkins UniversityORCID 0000-0003-0373-2816Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2

Frequent Co-Authors

Dan Wu· 2 shared Roger H. ReevesiD· 2 shared Yicong Li· 2 shared Alena Savonenko· 1 shared Alena V. SavonenkoiD· 1 shared Anna J. MoyeriD· 1 shared Atsuo OguraiD· 1 shared Atsushi Hasegawa· 1 shared

Activity by Year

2019-2021 · peak 1/yr

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10 top collaborators · 35 mutual co-author links

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2 shared papersNeuroscience

Dan Wu

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2 papers

Recent·Most cited

NeuroscienceActa Neuropathologica CommunicationsPeer-reviewedAug 16, 2021

Forebrain Shh overexpression improves cognitive function in a Down syndrome mouse model and euploid littermates

Feng Gao, Donna K. Klinedinst, Fabian‐Xosé Fernandez…·Roger H. Reeves

This study shows that overexpressing Shh in the forebrain of Down syndrome mice improves cognitive function, while cerebellar Shh overexpression does not.

14 citations

mouseexperimental assay

𝕏 PostBluesky

GeneticsbioRxivPreprintDec 10, 2019

A non-mosaic humanized mouse model of Down syndrome, trisomy of the long arm of human chromosome 21 in mouse chromosome background

Yasuhiro Kazuki, Feng J. Gao, Yicong Li…·Roger H. Reeves

This paper describes a new non-mosaic humanized mouse model of Down syndrome with a nearly complete copy of the long arm of human chromosome 21.

4 citations

mouseexperimental assay

𝕏 PostBluesky