Eva Kosek

The paper compares offset analgesia and onset hyperalgesia responses between women with non-suicidal self-injury and healthy controls, finding weaker onset hyperalgesia in the NSSI group.

This study examines skin neuroimmune changes in fibromyalgia patients, identifying novel dermal alterations and correlations between immune cell populations and nerve fiber density.

The paper examines lipid metabolism and anti-satellite glial cell IgG antibodies in fibromyalgia patients, finding associations between lipid concentrations, symptom severity, and autoantibody levels.

The paper examines bile acid and short-chain fatty acid levels in fibromyalgia patients and healthy controls, finding higher bile acid levels in patients and associations with autoantibodies and mental well-being.

This study finds that fibromyalgia patients with higher anti-satellite glia cell IgG antibodies have more severe symptoms, suggesting these antibodies may contribute to disease severity.

The paper identifies LPC16:0 as a mediator of chronic joint pain through ASIC3, supported by preclinical and clinical evidence.

This study shows that fibromyalgia pain is caused by IgG autoantibodies that sensitize sensory neurons in mice, suggesting potential treatment through reducing IgG levels.

This study evaluates the accuracy and reliability of specific binding estimates for [11C]PBR28 using the SIME method, showing good precision and reliability for VS but variability in BPND.