Author

Ulku Cuhadar

Centre National de la Recherche ScientifiqueORCID 0000-0002-9444-4133Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:differential expression· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Abhi AggarwaliD· 1 shared Alexandra JurczakiD· 1 shared Aman S. AberraiD· 1 shared Andreas GöebeliD· 1 shared Andreas Ritzau‐JostiD· 1 shared Angelika Sandstrom· 1 shared Azar Baharpoor· 1 shared Camilla I. SvenssoniD· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Abhi AggarwalORCID

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2 papers

Recent·Most cited

Activity-driven synaptic translocation of LGI1 controls excitatory neurotransmission

Ulku Cuhadar, Lorenzo Calzado-Reyes, Carlos Pascual-Caro, Aman S. Aberra, Andreas Ritzau‐Jost, Abhi Aggarwal, Keiji Ibata, Kaspar Podgorski, Michisuke Yuzaki, Christian Geis, Stefan Hallermann, Michael B. Hoppa, Jaime de Juan‐Sanz

This study shows that neuronal activity drives the translocation of LGI1 and ADAM23 to the synaptic cleft, adjusting excitatory neurotransmission based on activity history.

Cell Reports · Jan 1, 2022Peer-reviewedNeuroscience

Passive transfer of fibromyalgia pain from patients to mice.

Andreas Göebel, Clive Gentry, Ulku Cuhadar, Emerson Krock, Nisha Vastani, Serena Sensi, Katalin Sándor, Alexandra Jurczak, Azar Baharpoor, Louisa Brieskorn, Carlos Eduardo Morado-Urbina, Angelika Sandstrom, Janette Tour, Diana Kadetoff, Eva Kosek, , David A. Andersson

This study shows that fibromyalgia pain is caused by IgG autoantibodies that sensitize sensory neurons in mice, suggesting potential treatment through reducing IgG levels.

bioRxiv · Jul 24, 2019 · PreprintNeuroscience