Author

David A. Andersson

King's College LondonORCID 0000-0001-7451-8548Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:differential expression· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1

Frequent Co-Authors

Andreas GöebeliD· 2 shared Clive GentryiD· 2 shared Nisha VastaniiD· 2 shared Stuart BevaniD· 2 shared Alexandra JurczakiD· 1 shared Andrew TinkeriD· 1 shared Angelika Sandstrom· 1 shared Anne WorthingtoniD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersImmunology

Andreas GöebelORCID

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4 papers

Recent·Most cited

Pathways regulating the excitability of sympathetic neurons derived from human induced pluripotent stem cells

Laura Fedele, Margot Maurer, Andrew Tinker, David A. Andersson

This paper uses hiPSC-derived sympathetic neurons to study pathways regulating neuronal excitability and finds that GIRK channels mediate inhibitory effects of adrenergic and muscarinic receptors.

bioRxiv · May 27, 2026 · PreprintNeuroscience

The sensitising effect of IgG in fibromyalgia syndrome is mediated by Mrgprb2 in mast cells

Karla R. Sanchez, Jamie Burgess, Qin Zheng, Uazman Alam, Harvey Neiland, Richard Berwick, David A. Andersson, Samantha K. Korver, Anne Worthington, Andreas Göebel, Xinzhong Dong

This study shows that IgG in fibromyalgia syndrome activates mast cells via Mrgprb2, leading to hypersensitivity and inflammation.

bioRxiv · May 15, 2025 · PreprintImmunology

TRPA1 analgesia is mediated by kappa opioid receptors

Evangelia Semizoglou, Clive Gentry, Nisha Vastani, Cheryl L. Stucky, David A. Andersson, Stuart Bevan

This study shows TRPA1 inhibition enhances KOR activity in sensory neurons, leading to analgesia, and identifies TRP channel regulation of GPCR activity as a novel mechanism for peripherally mediated pain relief.

bioRxiv · Sep 1, 2022 · PreprintNeuroscience

Passive transfer of fibromyalgia pain from patients to mice.

Andreas Göebel, Clive Gentry, Ulku Cuhadar, Emerson Krock, Nisha Vastani, Serena Sensi, Katalin Sándor, Alexandra Jurczak, Azar Baharpoor, Louisa Brieskorn, Carlos Eduardo Morado-Urbina, Angelika Sandstrom, Janette Tour, Diana Kadetoff, Eva Kosek, , David A. Andersson

This study shows that fibromyalgia pain is caused by IgG autoantibodies that sensitize sensory neurons in mice, suggesting potential treatment through reducing IgG levels.

bioRxiv · Jul 24, 2019 · PreprintNeuroscience