Camilla I. Svensson

This study tests if netrin-1 inhibition reduces cancer-induced bone pain in three models but finds no significant anti-nociceptive effect.

This study identifies surface markers for isolating sensory neurons and develops a FACS panel for their enrichment without transgenic mice, validated by microscopy and scRNA-seq.

The paper develops a tissue-to-analysis framework to map the cellular and architectural organization of human dorsal root ganglia using transcriptomic and protein data.

This study identifies SEMA4D and OSM as mediators of neutrophil-neuron communication in arthritis-induced pain, showing their role in neuronal sprouting and hypersensitivity.

This study examines skin neuroimmune changes in fibromyalgia patients, identifying novel dermal alterations and correlations between immune cell populations and nerve fiber density.

The paper examines lipid metabolism and anti-satellite glial cell IgG antibodies in fibromyalgia patients, finding associations between lipid concentrations, symptom severity, and autoantibody levels.

The paper examines bile acid and short-chain fatty acid levels in fibromyalgia patients and healthy controls, finding higher bile acid levels in patients and associations with autoantibodies and mental well-being.

This study shows that androgen exposure in a mouse PCOS model alters immune cell populations in reproductive and metabolic tissues, with effects mediated through androgen receptors.

This study finds that fibromyalgia patients with higher anti-satellite glia cell IgG antibodies have more severe symptoms, suggesting these antibodies may contribute to disease severity.

The paper identifies LPC16:0 as a mediator of chronic joint pain through ASIC3, supported by preclinical and clinical evidence.

This study shows osteoclasts contribute to pain in arthritis by affecting bone vascularization and innervation, and inhibitors can reverse this pain.

This study shows that fibromyalgia pain is caused by IgG autoantibodies that sensitize sensory neurons in mice, suggesting potential treatment through reducing IgG levels.

The paper develops a cyclic smFISH method to map cell-specific markers in the somatosensory cortex, revealing its spatial organization using markers from single-cell transcriptomics.