Author

Liankun Gu

Peking University Cancer HospitalORCID 0000-0003-0791-1280Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Dajun DengiD· 3 shared Baozhen ZhangiD· 2 shared Hongfan YuaniD· 2 shared Sisi Qin· 2 shared Wei TianiD· 2 shared A‐Bin YouiD· 1 shared Chenghua Cui· 1 shared Jiaoyang Liu· 1 shared

Activity by Year

2019-2025 · peak 1/yr

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10 top collaborators · 30 mutual co-author links

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3 shared papersMolecular Biology

Dajun DengORCID

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3 papers

Recent·Most cited

Molecular BiologybioRxivPreprintJul 20, 2025

No phenotypes in Ttc22 knockout mice

Jiaoyang Liu, Liankun Gu, A‐Bin You…·Dajun Deng

This paper investigates the effects of Ttc22 knockout in mice and finds no significant phenotypes in development, fertility, or colon cancer induction.

DOI 10.1101/2025.07.20.665798

mouseproteomics

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Molecular BiologybioRxivPreprintMar 23, 2020

T606-phosphorylation deprives the function of Kaiso as a transcription and oncogenic factor

Wei Tian, Hongfan Yuan, Sisi Qin…·Dajun Deng

This study shows that AKT1 phosphorylates Kaiso at T606, promoting its cytoplasmic localization and de-repression of CDH1, with reduced phosphorylation linked to gastric cancer progression.

DOI 10.1101/2020.03.23.003509

humanexperimental assay

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Molecular BiologybioRxivPreprintApr 24, 2019

Hydroxymethylated-P16 Allele Is Transcription-Inactive

Ying Gan, Paiyun Li, Xiao Han…·Dajun Deng

This study shows that hydroxymethylated P16 alleles are transcriptionally inactive, demonstrated through engineered P16-specific dioxygenase-induced hydroxymethylation and its effects on cell growth and migration.

1 citation

humanDNA methylation

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