Young‐Yon Kwon

This study identifies sustained glucose turnover flux as a distinguishing metabolic feature of cancer cachexia compared to nutrient limitation under controlled food intake.

This study investigates the extent to which anorexia contributes to different cachectic phenotypes in cancer cachexia using a preclinical model.

The paper develops an organism-level quantitative flux model of energy metabolism in mice by integrating isotope tracing and mass spectrometry, revealing distinct metabolic cycling and oxidation flux patterns for circulating nutrients.