Author

Artur Veloso

ORCID 0000-0003-1735-7711Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Alejandro Álvarez-QuilóniD· 2 shared Jordan T.F. Young· 2 shared Julian Bowlan· 2 shared Li Li· 2 shared Michal ZimmermanniD· 2 shared Nancy LaterreuriD· 2 shared Stephen J. Morris· 2 shared Abira Rajah· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

Alejandro Álvarez-QuilónORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJan 25, 2026

CRISPR-enabled genetic screens identify synthetic lethal targets across frequently altered cancer drivers

Jessica Desjardins, Julian Bowlan, Cynthia Bernier…·Jordan T.F. Young

This study identifies synthetic lethal interactions between cancer drivers and tractable targets using CRISPR screens and cancer dependency data, offering new drug targets for therapy.

DOI 10.64898/2026.01.25.701589

humangenetic perturbation / CRISPR

𝕏 PostBluesky

Cancer BiologybioRxivPreprintFeb 15, 2025

A dual mechanism of sensitivity to PLK4 inhibition by RP-1664 in neuroblastoma

Isabel Soria‐Bretones, Matias Casás‐Selves, Elliot Goodfellow…·Michal Zimmermann

This study shows neuroblastoma cells are hypersensitive to PLK4 inhibitor RP-1664 due to dual mechanisms of centriole loss and amplification, leading to cell death.

5 citations

humanbulk RNA-seq

𝕏 PostBluesky