William K. Russell

The paper investigates cellular responses to gas vesicle collapse using transcriptomic and proteomic analyses in native and engineered bacterial systems, revealing distinct stress responses and no clear protein recognition mechanism.

The paper shows QKI competes with SF1 for branch point sequences, repressing splicing and causing lethality in yeast.

The paper identifies RNA-protein modules linked to anthracycline-induced cardiotoxicity and atrial fibrillation risk through multi-omics integration.

This study identifies REEP2 as a key mediator of EMT-driven membrane trafficking in lung cancer, promoting metastasis through enhanced ER-to-Golgi transport.

This study shows that cell-free amniotic fluid reduces liver fibrosis by inhibiting hepatic stellate cell activation in multiple models.

The paper identifies DNA damage-associated protein co-expression modules in cardiomyocytes and shows their relationship to genetic variation tolerance and CVD risk.

This study identifies how NITD-688 inhibits dengue virus by binding NS4B and disrupting its interaction with NS3, reducing viral replication.

This study identifies a mechanism where HTT recruits BRG1 and NHEJ components to repair transcription-associated DSBs in neurons, and shows that mutant HTT impairs this process, contributing to HD pathology.

The paper identifies an atlas of 3QTLs associated with alternative polyadenylation across 46 tissues, showing they explain 16.1% of trait-associated variants and contribute to disease independently of gene expression.

The paper identifies alternative splicing programs in three germ layers and shows Quaking's role in cardiac mesoderm specification through splicing regulation.