William Havelange

This study shows that rbfox1 loss of function in zebrafish disrupts stress-related gene expression and increases cortisol levels during development, leading to behavioral and physiological signs of allostatic overload in adulthood.

The paper generates zebrafish mutants with fzd9b disruptions to study anxiety and social behaviors linked to Williams syndrome, finding altered stress responses and compensatory gene upregulation.

This study examines the behavioral effects of developmental JWH-018 exposure in zebrafish and finds no moderation by disc1 mutations in anxiety-like behaviors.