Author

Maria Sara Remedi

Washington University in St. LouisORCID 0000-0003-2048-472XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Organism:human· 1

Frequent Co-Authors

Caitlin M. CarrolliD· 2 shared Celeste M M. KarchiD· 2 shared Colin G. NicholsiD· 2 shared David M. HoltzmaniD· 2 shared Emily E. CaesariD· 2 shared James Andy Snipes· 2 shared John Grizzanti· 2 shared Molly StanleyiD· 2 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersNeuroscience

Caitlin M. CarrollORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintFeb 20, 2022

Kir6.2-containing KATP channels are necessary for glucose dependent increases in amyloid-beta and Alzheimer's-related pathology

John Grizzanti, William R. Moritz, Morgan C. Pait…·Shannon L. Macauley

This study shows that Kir6.2-KATP channels are necessary for hyperglycemia-induced increases in amyloid-beta and Alzheimer's pathology by regulating lactate transport and Aβ release.

2 citations

humanmouse

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NeurosciencebioRxivPreprintAug 11, 2021

Sulfonylureas target the neurovascular response to decrease Alzheimer's pathology

Shannon L. Macauley, Molly Stanley, Emily E. Caesar…·David M. Holtzman

This study shows that sulfonylureas reduce Alzheimer's pathology by targeting vascular KATP channels, decreasing amyloid-beta aggregation and improving vascular function in mice.

4 citations

mouseexperimental assay

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