Molly Stanley

This study shows that food perception alone, through olfactory signaling and serotonin receptors, stimulates oviposition in Drosophila without requiring nutrient intake.

This study shows that insulin receptor signaling in sweet gustatory receptor neurons of Drosophila modulates sugar taste sensitivity and feeding behavior based on metabolic state.

This study shows artificial sweeteners activate both sweet and bitter taste neurons in Drosophila, with varying behavioral effects depending on concentration and compound type.

This study identifies the insulin/insulin-like growth factor signaling pathway as a key determinant of higher fat storage in Drosophila females through increased dietary sugar intake and Dilp3 expression.

This study identifies taste cells and receptors in Drosophila that mediate amino acid attraction and aversion, revealing parallel pathways for AA detection.

The paper uses connectome and functional imaging data to show taste circuits in Drosophila are organized with segregated taste representations in specific interneurons and projection neurons.

This study identifies IR94e-expressing gustatory receptor neurons in Drosophila that suppress feeding and enhance egg laying in response to amino acids.

This study shows that acute hyper- and hypoglycemia disrupt the metabolic cooperation between glucose and lactate in the brain, affecting sleep-wake cycles.

The paper identifies IR7c as a key receptor for high salt taste in Drosophila, showing it works with co-receptors to detect cation non-selective salts and can convert sodium-selective receptors to non-selective ones.

This study shows that Kir6.2-KATP channels are necessary for hyperglycemia-induced increases in amyloid-beta and Alzheimer's pathology by regulating lactate transport and Aβ release.

This study shows that sulfonylureas reduce Alzheimer's pathology by targeting vascular KATP channels, decreasing amyloid-beta aggregation and improving vascular function in mice.

The paper shows lactic acid is an appetitive taste in Drosophila, activating sweet GRNs through two distinct receptor-mediated phases.

This study shows that reducing olfactory input in Drosophila increases their sensitivity to both appetitive and aversive tastes, suggesting a homeostatic mechanism compensating for sensory loss.