Gopal V. Velmurugan

This study examines sex-specific mitochondrial changes in a mouse model of traumatic brain injury, revealing distinct spatial and temporal responses in mitochondrial function across brain regions.

This study shows astrocytes transfer mitochondria to neurons after TBI to support mitochondrial function in neuronal soma, dendrites, and axons but not synapses, and demonstrates therapeutic potential of exogenous mitochondrial transfer via extracellular vesicles.

This study investigates how tau pathology affects metabolism and excitatory activity in mouse models, finding that it preserves glucose tolerance and shifts metabolic flux toward glutamate synthesis.